FNH-like nodules: Possible precursor lesions in patients with focal nodular hyperplasia (FNH)

Comparative Hepatology

Table 2 Data of cases

Cases	Age	Discovery	Other findings	Surgery	Pathological findings of non nodular liver
1	37	Abdominal pain	Type 1 diabetes, Hashimoto thyroiditis	LH	Some foci of steatotic and clear hepatocytes. Mildly enlarged portal tracts with thickened arteries.
2	49	Abdominal pain	Gallbladder lithiasis	LH	Micro and macro steatosis (30 %). Few foci of steatotic hepatocytes, some portal tracts with enlarged arteries.
3	35	Incidental (episode of fever and rise in transaminases)	Persistant rise in GGT	RH	Macrovesicular steatosis (15 %). Small and rare peliotic areas. Accentuation of the lobular septation.
4	39	Incidental		LH	Macrovesicular steatosis (10 %). Small and rare peliotic areas. Some portal tracts with enlarged arteries.
5	49	Follow up for chronic alcoholism		LH + IV	Severe steatosis (60 %). Few limited areas with dilated sinusoids and peliosis. Often enlarged arteries.
6	29	Incidental		VII
7	32	Abdominal pain	Hemangioma	V, VI	Around nodule b, approximation of portal tracts, absence of portal veins, thickened wall of hepatic veins, one steatotic focus.
8	47	Abdominal pain		V, VI	Macrovesicular steatosis (50 %) medio/centro lobular.

Cases 1 to 7 are women. Roman numbers indicate the segments of the liver. GGT: gammaglutamyl transpeptidase; HCC: hepatocellular carcinoma: LH: left hepatectomy; LL: left lobe; RL: right lobe.